Nicotinamide adenine dinucleotide (NAD+) is a coenzyme used by the body which is fundamental for energy metabolism. In addition, NAD+ also plays multiple direct and indirect roles in cellular regulation and functions such as:
- DNA repair
- Protection against oxidative stress
- Slows cellular damage due to aging
- Reduces inflammation and improves immune cellular function
A link to NAD+ levels and health was made as far back as 1937 and is still being studied to this day for its link to neurodegenerative and metabolic diseases as well as its correlation to aging.
As someone ages their NAD+ levels also decline which have been linked to aging related diseases such as:
- Atherosclerosis
- Arthritis
- Hypertension
- Cognitive decline
- Diabetes
- Cancer
David Sinclair of the Paul F. Glenn Center for the Biology of Aging at Harvard Medical School stated that NAD+ is “the closest we’ve gotten to a fountain of youth.”
Clearly, NAD+ plays an essential role within the body and its various functions. As NAD+ levels decline, increased chances of disease and the effects of aging may arise. Therefore, increasing levels of NAD+ can prove beneficial for lasting health and wellness.
Ways to boost NAD+ levels:
- Lifestyle changes
- Diet, exercise, eating a healthy diet, consistent and quality sleep
- Supplementation
- Oral precursors (nicotinamide mononucleotide and nicotinamide ribosome)
- IV Infusions
At Nova Vita Wellness Centers, our NAD+ infusion will help increase dwindling NAD+ levels in a safe and efficacious manner and provide your body with the tools it needs to function at its best.
Book an appointment online today and discover the benefits of NAD+ for yourself.
References:
- Covarrubias, Anthony J., et al. “NAD+ metabolism and its roles in cellular processes during ageing.” Nature Reviews Molecular Cell Biology 22.2 (2021): 119-141.
- Lautrup, Sofie, et al. “NAD+ in brain aging and neurodegenerative disorders.” Cell metabolism 30.4 (2019): 630-655.
- Yang B, Dan X, Hou Y, et al. “NAD+ supplementation prevents STING-induced senescence in ataxia telangiectasia by improving mitophagy.” Aging Cell. (2021)
